CHAPTER 11 Clinical Treatment
DEFINITION OF DENTAL HYGIENE PRACTICE
Dental hygiene is the science and practice of recognition, treatment, prevention of oral diseases. Hygienist is a preventive oral health professional who has graduated from an accredited program in an institution of higher education; a licensed professional who provides educational, clinical, research, administrative, therapeutic services supporting total health through promotion of optimal oral health. In practice, hygienists integrate roles of clinician, educator, advocate, manager, researcher to prevent oral diseases and promote health; hygienists work in partnership with dentists.
• See CD-ROM for ADHA Guidelines for the Standards For Clinical Dental Hygiene Practice (SCDHP) (2008).
A. Process of care consists of six components (ADPIED):
1. Assessment: systematic collection, analysis, documentation of the oral and general health status and patient needs.
2. Dental hygiene diagnosis: component of the overall dental diagnosis; identification of existing or potential oral health problem that a dental hygienist is educationally qualified and licensed to treat.
3. Planning: establishment of goals and outcomes based on patient needs, expectations, values, current scientific evidence.
4. Implementation: delivery of dental hygiene services based on dental hygiene care plan in a manner minimizing risk and optimizing oral health.
B. Dental hygiene process of care is a cycle in which the hygienist might pass through each of the recommended steps (ADPIED) several times during a course of treatment; over a period of months or years, a hygienist may have evaluated the process several times, altering the diagnosis and plan numerous times as the patient’s condition changes.
DENTAL RECORD
• See Chapters 16, Special Needs Patients: medical disabilities; 18, Ethics and Jurisprudence: HIPAA, informed consent.
Health History
• See Chapters 6, General and Oral Pathology: common conditions and diseases in dental setting; 8, Microbiology and Immunology: vaccinations, infectious diseases; 9, Pharmacology: antibiotic premedication; 10, Medical and Dental Emergencies: medical history, physical status classification (ASA), high blood pressure.
A. Medical history: review of physical health status.
2. Need for medical consult and antibiotic and/or antianxiety premedication require special precautions or antibiotic premedication MUST be assessed.
3. Information obtained by:
a. Questionnaire vs. interview:
(1) Questionnaire: saves time, consistent, potentially MORE thorough; however, inflexible and LACKS opportunity for development of rapport with patient; impersonal.
B. Health status: notation of existing and previous conditions.
1. Includes chief complaint (CC) and formation of physical classification status (American Society of Anesthesiologists [ASA]):
c. History of present illness; may influence future treatment options and setting.
d. History of past illness includes:
C. Medically compromised patients who can be seen in a dental setting but need major modification of dental treatment and/or need medical consult (drug history discussed next):
1. Patient with cardiovascular disease (CVD):
a. Has inadequate blood circulation to heart muscle, resulting from arteriosclerosis, blocking or narrowing of blood vessels; MUST be ready for an emergency.
b. Possibly reports angina pectoris caused by muscle pain that radiates to left arm and jaw; experiences pain with exertion and anxiety; discomfort is relieved by rest and vasodilator nitroglycerin (NTG, Nitrostat); request that patient bring NTG to dental appointment; have readily available during treatment.
c. May use antihypertensives (includes diuretics, adrenergic blockers) that may result in xerostomia.
d. Patient after myocardial infarction (MI, heart attack): attack resulted from insufficient blood supply to heart muscle caused by atherosclerosis of blood vessel walls; after cardiac episode and/or surgery, requires 4- to 6-week delay before emergency or elective dental procedures.
2. Patient with hypertension (high blood pressure [HBP]): has or had elevated blood pressure; may also report symptoms of headache, dizziness, or nosebleeds:
b. Can progress to arteriosclerosis, impaired renal function, cardiac enlargement, MI, cerebrovascular accident (CVA, stroke); MUST be ready for an emergency.
3. Patient with congestive heart failure (CHF): occurs when heart muscle is weak and is unable to pump blood at an adequate rate; blood circulation is poor, resulting in congestion and pooling of blood in the organs and lower extremities.
4. Patient with cardiac dysrhythmia: had or has irregular heartbeat (too slow or fast).
a. May have pacemaker (with or without defibrillator) and report taking digitalis glycoside (digoxin, Lanoxin) or calcium channel blocker such as diltiazem (Cardizem, Dilacor, Tiazac) or nifedipine (Procardia); may have xerostomia and gingival hyperplasia.
5. Patient with valvular heart disorder:
6. Patient with diabetes mellitus (DM): has disorder of glucose metabolism resulting from relative or absolute lack of insulin; has vascular component (may have related CVD and/or renal diseases; see later discussion).
a. Two types: type 1, can be severe and unstable; stems from lack of insulin produced from pancreas, and type 2, often NOT as severe but usually stable; develops slowly with age; frequently associated with obesity; pancreas produces adequate insulin but there is insulin insensitivity of the tissue.
b. Dental appointment should be scheduled after meal and insulin therapy if taken; MUST be ready for emergency.
7. Patient with renal disease: renal function is impaired at various levels, possibly maintained by hemodialysis or kidney transplant.
8. Patient with blood disorder or with history of difficulty with bleeding:
a. Patients include those with anemia, deficiency of red blood cells (RBCs) resulting from vitamin or iron deficiency, bone marrow malfunction, excessive loss of blood, or RBC destruction.
b. Also include patients with leukemia, cancer of white blood cells that do NOT function normally (cells are immature and excessive in number); thrombocytopenia may develop from chemotherapeutic treatment.
9. Patient with respiratory infection or disease:
10. Patient with sexually transmitted disease or infection (STD, STI):
11. Patient with hepatitis A, B, C, D, E: patients ALL have inflammation of the liver resulting from viral infection; causes fatigue, nausea, tender joints, jaundice.
a. Follow recommendations for hepatitis B vaccination for ALL dental providers before treating patients in any clinical setting, since carrier status is frequently unknown.
12. Patient with allergy: can have reactions to substance that may be as slight as a skin rash or as severe as fall in blood pressure and anaphylactic shock (airway obstruction from tissue swelling and/or cardiac arrest).
a. Reports reaction to known substance (allergen) such as antibiotics, rubber latex, restorative metals (especially nickel), other allergens; need to AVOID allergen.
b. May have asthma; need to recommend acetaminophen as substitute for its analgesic and antipyretic effects to avoid aspirin-induced attack; may need to alter local anesthetics (AVOID vasoconstrictor or articaine).
13. Patient with epilepsy: patient can have disturbance of electrical brain activity resulting in seizure (convulsions).
Drug History
• See Chapters 6, General and Oral Pathology: specific conditions and diseases noted in dental setting; 9, Pharmacology: drugs that affect dental care.
A. Pharmacological record: information regarding drug action, usage, contraindications, adverse reactions, warnings, precautions is reviewed.
1. Includes record of drugs taken (including over the counter [OTC]), noting dosage and condition being treated; note if drug has impact on oral health and any contraindications for dental treatment.
2. Common drug-induced oral side effects:
a. Xerostomia (dry mouth): associated with cracked lips, sore labial commissures, inflamed smooth tongue (glossitis).
(1) MOST common reaction to drugs; MOST commonly to drugs used to treat depression and anxiety, urinary incontinence, Parkinson’s disease, as well as antihistamines, antihypertensives, antidiarrheals, muscle relaxants; BOTH prescription and OTC, such as for colds, flu, allergies.
(2) Results in thick and ropy saliva, altered taste, loss of buffering action; may be associated with burning tongue.
(3) Increases incidence of caries, especially of the root; causes difficulty with chewing, swallowing, speaking, bad breath, denture wearing.
(4) Treatment: relieving symptoms through increased water consumption (sip water), saliva substitutes, tissue lubricants, chewing sugar-free gum (especially with xylitol), air humidification; AVOID alcohol mouthrinses that can dehydrate tissues; to prevent caries, use of home fluoride applications and calcium products for additional remineralization; see earlier discussion if non–drug related.
b. Candidiasis: overgrowth of yeast organism, Candida albicans (see Chapter 8, Microbiology and Immunology):
(1) Increased risk with history of prolonged antibiotics, immunosuppressants, or corticosteroid therapy with immunocompromised health, denture or dental appliance wear.
c. Drug-induced gingival hyperplasia: associated with enlarged, bulbous, and fibrotic gingival tissues:
(1) Results from anticonvulsant phenytoin (Dilantin), calcium channel blocker such as nifedipine (Procardia), or transplant rejection drugs such as cyclosporine.
3. Common drugs that affect dental treatment:
a. Antihypertensives: used to reduce high blood pressure (HBP) with cardiovascular disease (CVD).
(1) Can cause orthostatic (postural) hypotension (reduced blood pressure): sit patient up slowly and keep seated for several minutes to reduce dizziness of hypotension:
(4) Nifedipine (Procardia), calcium channel blocker, can cause gingival hyperplasia (discussed earlier).
b. Diuretics: used to promote renal excretion in the treatment of CHF and HBP:
c. Anticoagulants and antiplatelets: used for treatment of CVD to increase blood flow by suppressing or delaying coagulation of blood:
d. Antianginals: used for treatment of angina pectoris to increase oxygen supply to heart muscle through vasodilation:
e. Cardiac drugs:
g. Vasoconstrictors: used to alleviate symptoms of asthma and emphysema; taken as inhalants.
(1) Can cause xerostomia (see earlier discussion); frequently associated with inflamed gingival tissues because of mouth breathing (associated with asthma).
i. Antiinflammatory drugs: depress inflammatory response and treat adrenocortical insufficiency, rheumatoid arthritis (RA), and respiratory disease (emphysema):
j. Antidepressants: taken for mental disorders, weight loss, tobacco cessation, sleep disorders, temporomandibular disease (TMD).
(1) May cause xerostomia (see earlier discussion) and taste alterations; may cause bruxism with resultant attrition.
PATIENT ASSESSMENT
Patient Examination
• See Chapters 4, Head, Neck, and Dental Anatomy: head and neck anatomy; 6, General and Oral Pathology: diagnosis.
A. Examination technique: includes visual observation, palpation, auscultation, olfaction.
1. Visual observation, BOTH direct and indirect (mouth mirror), to examine intraoral and extraoral structures:
2. Palpation used to examine tissues and underlying structures:
b. Bidigital: tissue grasped between finger(s) and thumb (cheeks, alveolar ridges, lips, vestibule, tongue, ducts, floor of mouth, larynx).
c. Manual: all fingers of one hand used to grasp tissue (anterior deep and superficial cervical nodes).
d. Bimanual: finger(s) and thumb from each hand applied simultaneously to examine tissues (floor of the mouth).
Extraoral Examination
A. Overall physical appearance by visual examination:
1. Unsteadiness of gait: CANNOT walk at a normal pace and maintain balance because of orthopedic disabilities or influence of drugs.
2. Restricted mobility: limps while walking, which may indicate injury to hip, leg, knee, or foot or systemic disease; investigate possible surgery that required pins, joint repair, or prosthesis.
3. Imbalance: CANNOT walk without losing balance; may be due to equilibrium problem, inner ear infection, loss of muscular strength, or damage from CVA.
B. Hair by visual examination and palpation:
D. Skin by visual examination and palpation:
E. Eyes by visual examination and reaction to light source:
G. Ears by visual examination and palpation:
H. Bones, muscles, lymph nodes, glands using visual and palpation:
1. Parotid glands: bilateral circular compression for pain, swelling, enlargement, hardness; salivary flow observed at opening of duct across from maxillary first molar on buccal mucosa when gland is milked.
2. TMJ: bilateral palpation with BOTH index fingers slightly anterior to outer meatus, have patient open and close several times, slowly; note pain, clicking, popping, grinding, and restriction in opening or closing; note any deviations, including during patient interview; may have TMD.
3. Masseter and temporalis muscles: bilateral circular compression for pain, swelling, enlargements, unusual hardness; masseter may be enlarged because of bruxism and/or clenching habits.
4. Border of mandible: bimanual palpation from midline to posterior angle for changes in contour and pain.
5. Mentalis muscle: digital palpation, rolling tissue over mandible, for smoothness or restriction in swallowing movement.
6. Lymph nodes: assessed for tenderness or pain, swelling, enlargement, unusual hardness, fixed position:
c. Superficial cervical (submental, submandibular, anterior and external jugular): digital palpation with fingers anteriorly from midline of mandible with head down to posteriorly to angle of mandible, rolling tissue over jaw, and then down to and along sternocleidomastoid muscle (SCM), with patient’s head to side.
7. Submental and submandibular salivary glands: bilateral digital palpation for asymmetry, noncontiguous borders, pain, swelling, enlargement, unusual hardness, difficulty in swallowing.
9. Thyroid gland: displacing gland to one side of neck, then combination of bimanual and manual palpation inferolaterally to cartilage (“Adam’s apple”) while patient sits upright; ask patient to swallow (may need glass of water) to check for mobility; note asymmetry, nodules, enlargement (goiter), or surgical removal.
Intraoral Examination
A. Lips by visual examination and bidigital palpation:
B. Labial and alveolar mucosa using visual examination and bidigital palpation:
2. Tight frenum attachments: can cause gingival defects such as loss of attached gingiva (mucogingival defect).
5. Hyperkeratosis: use of lozenges, drugs, or spit (smokeless) tobacco products held in mandibular vestibule; careful evaluation of changes is essential.
6. Smokeless (spit) tobacco lesion: hyperkeratinized tissue; white, sometimes corrugated, in appearance.
C. Buccal mucosa using visual examination and palpation:
1. Check parotid (Stensen’s) duct using bilateral palpation: pain, enlargements, tumors, calcified areas; milk the ducts to confirm function.
D. Gingiva using visual examination and palpation:
2. Infection, with redness and swelling (inflammation), such as McCall’s festoons (lifesaver marginal gingiva): inadequate dental biofilm removal and presence of periodontal disease, mainly gingivitis, possibly periodontitis.
4. Exostosis (plural, exostoses): small benign bone growths projecting from alveolar process, MAINLY on maxillary facial surface.
E. Hard and soft palate using visual inspection and digital palpation:
1. Torus (plural, tori): hard protruding bony structure (benign exostosis), NOT of significance unless partial or full denture is being constructed, then may need to be reduced or removed.
2. Ulcerations: fluid-filled lesions surrounded with red halo with aphthous ulcers, burns, viral infection, or autoimmunity (broken blisters).
3. Trauma: mechanical or chemical irritation; should heal within 10 days, if not, further investigation needed to rule out pathological condition or child abuse.
4. Stomatitis: ranges from small, red, petechia-like lesions (nicotine and denture stomatitis) to generalized and granular (denture stomatitis) to ulcerative (aphthous stomatitis) to intense redness with focal bone loss (necrotizing stomatitis).
F. Floor of mouth using visual inspection and palpation:
1. Check submandibular (Wharton’s) duct: can become blocked with mucous plugs or sialoliths (present on occlusal radiograph).
G. Tongue using visual examination and palpation:
1. Coating: varying degree of keratinization of filiform lingual papillae; food, drugs, bacteria (halitosis); staining caused by tobacco use; tongue cleaning needed.
2. Fissuring: presence of numerous grooves and crevices on dorsal surface and lateral borders of tongue; entrap food and bacteria; tongue cleaning needed; MORE common with Down syndrome.
4. Lateral border and ventral surface: high-risk area for oral cancer, with red and/or white lesions, bleeding, ulceration.
5. Other common lesions of the tongue:
a. Ulcers: aphthous is MOST common, with systemic disease such as viral infections or autoimmunities or with smoking cessation.
c. Geographic: common benign condition involving filiform lingual papillae found on dorsum and borders; lesions are red areas with white borders; outline of lesions changes, heals, reappears in different area.
d. Median rhomboid glossitis (central papillary atrophy): flat or lump benign lesion on dorsum, NO filiform present; related to chronic candidiasis.
e. Tongue thrust (during swallowing): anterior portion pushes between teeth; ideally SHOULD be positioned on palate behind maxillary central incisors while swallowing; retraining may be necessary if thrusting causes speech disorders or tooth positioning problems (malposed teeth).
f. White plaques: lichen planus, candidiasis, or other systemic disease; definitive diagnosis is needed.
g. Black hairy tongue: elongation of filiform with dark staining: long-term use of certain drugs or tobacco; long-term rinsing with undiluted hydrogen peroxide.
CLINICAL STUDY
4. Are there any special treatment considerations because of the patient’s hemophilia or HIV status?
1. Hemophilias are a group of disorders involving blood-clotting mechanisms. Diseases are genetic, do not produce factor VIII (hemophilia A), factor IX (hemophilia B, or Christmas disease), factor IX (hemophilia C), or von Willebrand’s factor (vWF). Treatment includes coagulation therapy.
2. Additional information needed before treatment would include type and severity of hemophilia, treatment regimen for control of hemophilia, drugs being taken for hemophilia, HIV status including CD4 cell counts (lymphocyte subset cell count), presence of any AIDS-associated conditions (includes oral signs), and any drugs (both prescription and OTC) being taken to manage HIV condition. In addition, INR numbers would be needed to ascertain bleeding levels; medical consult needed. Antifibrinolytic agents (Cyklokapron) effectively prevent oral bleeding when they are combined with a preventive dose of clotting factor and can be used with oral surgery.
3. Hemophilia A, B, and C are rare in women; however, von Willebrand’s disease does occur in both men and women.
4. Standard precautions are followed for every patient. Light debridement (scaling) is only instrumentation needed in the patient’s case because of minimal deposits and inflammation; may want to avoid ultrasonic scalers and use manual instrumentation, since aerosols created may be a health risk for immunocompromised patient. However, he should be seen for preventive maintenance every 3 months because of high-risk status. Excellent oral hygiene self-care and preventive therapy reduce chances for oral infection and bleeding.
5. Before 1985, blood stores used for transfusions were not required to be tested for HIV virus, and many hemophiliacs contracted virus.
6. Thorough intraoral and extraoral examination is critical for noting progression of patient’s HIV status, since many early indicators of disease progression are exhibited in the mouth. Palpate for any persistent generalized lymphadenopathy (PGL) and visually observe for any skin lesions such as Kaposi’s sarcoma, purpura, or herpes. Check for common oral signs of progressing AIDS virus, su/>